Series of orthopaedic knowledge introduction (Ⅷ)

Last issue we learned about membranous bone and chondrogenic bone.

On this issue, we would like to talk about the plasticity of bone.

To be specific, osteoporosis occurs when neuroregulation is reduced. People with palsy disease may suffer from bone decalcification, osteoporosis and bone resorption due to nerve injury, and even spontaneous fracture. Endocrine has a great influence on the development of bone. Before adulthood, the secretion of multiplicator growth hormone will promote the excessive growth of bone, which leads to gigantism. If the division is insufficient, then development arrest leads to somatism. In adults, hypersecretion of growth hormone is associated with acromegaly. Vitamin A regulates the function of osteoblasts and osteoclasts and maintains the normal growth of bone. Vitamin D can promote the intestinal absorption of calcium and phosphorus. Deficiency of Vitamin D is associated with a decrease in calcium and phosphorus in the body, which affects bone calcification, causing disease in childhood and osteomalacia in adults.

After fracture, callus forms at the fracture site. At the initial stage of fracture healing, bone callus is irregular. After a certain period of absorption and reconstruction, the bone can basically restore the original morphology and structure.

All in all, the plasticity of bone plays a vital role in the initiation and growth of bones.

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